How to prevent and treat chronic diseases starts from protecting the intestinal tract
Summary: the beneficial bacteria in the intestinal tract play a great role in the body's immunity. If these beneficial bacteria and protective layers are destroyed, the inflammatory factors will become weak, and the prevention of chronic diseases must start from the protection of the intestinal tract.
Sacrifice the local to protect the whole inflammatory factors.
For a long time, the medical profession has reminded that the concentration of some key inflammatory factors in the blood of patients with chronic diseases such as obesity, diabetes and coronary heart disease is higher than that of ordinary people. These inflammatory factors are produced by immune cells and are used to destroy infected human cells, sacrifice parts, and protect the whole.
In general, the concentration of these inflammatory factors will only increase at the site of infection. In obese people, these inflammatory factors are scattered in the blood of the body, threatening the cells of the whole body. For example, when the concentration of tumor necrosis factor a (TNFa, inflammatory factor) increases, the structure of the insulin receptor in the cell changes, and the receptor cannot sense the insulin concentration signal in the blood. As a result, after an increase in blood glucose levels leads to a rise in insulin levels, these cells do not absorb sugar from the blood and reduce blood sugar levels. This is the early stage of diabetes, also known as insulin resistance.
The medical community has reached a consensus on metabolic diseases caused by chronic inflammation, but there is no consensus on the causes of chronic inflammation.
Pathogens increase high-fat foods.
Pathogens usually obtain nutrition by decomposing animal tissue. High-fat foods usually add a lot of fat. When the oil enters the large intestine, it nourishes the bacteria and increases the total amount of the bacteria.
While beneficial bacteria (such as Bifidobacterium) cannot use animal tissue to obtain nutrition, the nutrition they need is dietary fiber that the human body cannot digest, so they will not destroy animal tissue. Too much animal nutrition in food, too little food fiber, beneficial bacteria gradually reduce or disappear due to lack of nutrition, replaced by pathogens.
The endotoxin released by bacteria can enter the bloodstream. When leukocytes find endotoxin (that is, antigen) in the blood, they think that the pathogen invades and immediately produces inflammatory factors. High concentrations of inflammatory factors eventually destroy the healthy cells of animals. For a long time, some important functions have been destroyed, resulting in a variety of chronic diseases.
A journey to explore the causes of chronic inflammation.
Scientists from France, Britain and other countries have conducted in-depth studies on the causes of chronic inflammation in obese people and come to the conclusion that the structural imbalance of intestinal flora can cause chronic inflammation.
The first stop: feed the mice with high-fat diet and use the mice to carry out experimental research. First of all, the mice were fed with a high-fat diet, and the concentration of endotoxin in the blood of obese mice was higher than that of healthy control mice fed with normal diet. Endotoxin, also known as lipopolysaccharide, is a component of the cell wall of Gram-negative bacteria. If these ingredients enter the bloodstream, they stimulate white blood cells to secrete inflammatory factors. They then injected the purified endotoxin into healthy mice fed a normal diet, resulting in significant weight gain, chronic inflammation and insulin resistance. This suggests that even if mice eat a low-calorie diet big ass , they will be stimulated by chronic inflammation and become obese.
The second station: the receptor was used to beat the mouse to test endotoxin to stimulate white blood cells to produce inflammatory factors, which first combined with leukocyte receptor CD14. If the CD14 is destroyed, white blood cells cannot sense endotoxins in the blood or produce inflammatory factors. It was found that when endotoxin was injected into CD14 knockout mice rabbit vibrator , these mice did not produce an inflammatory response and therefore did not develop obesity and insulin resistance.
The third station: the intestinal flora of mice was analyzed to determine the increase of antigens in the intestines and the way of entering the blood. the scientists analyzed the structure of intestinal flora of experimental mice by fluorescence in situ hybridization (fish). The results showed that the intestinal flora structure of obese mice fed with high-fat diet changed, the number of some beneficial bacteria (such as Bifidobacterium) decreased significantly, while the number of some Gram-negative bacteria increased significantly. This suggests that high-fat diet can increase the number of intestinal pathogenic bacteria, and then increase the amount of intestinal antigen.
A large number of studies have found that Bifidobacteria mainly grow on the intestinal mucosa, they can form a biofilm and have a protective effect on the intestinal tract. Once these bacteria are reduced, the intestinal wall will be damaged and the permeability will increase, which is what people call the intestines "leaking". Intestinal leakage, fecal poison into the blood, will naturally cause chronic inflammation, and eventually lead to chronic diseases.
Station 4: scientists who fed enough oligosaccharides to experimental mice added sufficient amounts of bifid factor (oligosaccharides) to a high-fat diet. Although the number of intestinal pathogens increased, because the number of beneficial bacteria did not decrease, antigens could not enter the bloodstream and could not stimulate leukocyte inflammation. This strong evidence suggests that an unreasonable diet destroys intestinal flora, causing chronic inflammation and chronic diseases.
Sacrifice the local to protect the whole inflammatory factors.
For a long time, the medical profession has reminded that the concentration of some key inflammatory factors in the blood of patients with chronic diseases such as obesity, diabetes and coronary heart disease is higher than that of ordinary people. These inflammatory factors are produced by immune cells and are used to destroy infected human cells, sacrifice parts, and protect the whole.
In general, the concentration of these inflammatory factors will only increase at the site of infection. In obese people, these inflammatory factors are scattered in the blood of the body, threatening the cells of the whole body. For example, when the concentration of tumor necrosis factor a (TNFa, inflammatory factor) increases, the structure of the insulin receptor in the cell changes, and the receptor cannot sense the insulin concentration signal in the blood. As a result, after an increase in blood glucose levels leads to a rise in insulin levels, these cells do not absorb sugar from the blood and reduce blood sugar levels. This is the early stage of diabetes, also known as insulin resistance.
The medical community has reached a consensus on metabolic diseases caused by chronic inflammation, but there is no consensus on the causes of chronic inflammation.
Pathogens increase high-fat foods.
Pathogens usually obtain nutrition by decomposing animal tissue. High-fat foods usually add a lot of fat. When the oil enters the large intestine, it nourishes the bacteria and increases the total amount of the bacteria.
While beneficial bacteria (such as Bifidobacterium) cannot use animal tissue to obtain nutrition, the nutrition they need is dietary fiber that the human body cannot digest, so they will not destroy animal tissue. Too much animal nutrition in food, too little food fiber, beneficial bacteria gradually reduce or disappear due to lack of nutrition, replaced by pathogens.
The endotoxin released by bacteria can enter the bloodstream. When leukocytes find endotoxin (that is, antigen) in the blood, they think that the pathogen invades and immediately produces inflammatory factors. High concentrations of inflammatory factors eventually destroy the healthy cells of animals. For a long time, some important functions have been destroyed, resulting in a variety of chronic diseases.
A journey to explore the causes of chronic inflammation.
Scientists from France, Britain and other countries have conducted in-depth studies on the causes of chronic inflammation in obese people and come to the conclusion that the structural imbalance of intestinal flora can cause chronic inflammation.
The first stop: feed the mice with high-fat diet and use the mice to carry out experimental research. First of all, the mice were fed with a high-fat diet, and the concentration of endotoxin in the blood of obese mice was higher than that of healthy control mice fed with normal diet. Endotoxin, also known as lipopolysaccharide, is a component of the cell wall of Gram-negative bacteria. If these ingredients enter the bloodstream, they stimulate white blood cells to secrete inflammatory factors. They then injected the purified endotoxin into healthy mice fed a normal diet, resulting in significant weight gain, chronic inflammation and insulin resistance. This suggests that even if mice eat a low-calorie diet big ass , they will be stimulated by chronic inflammation and become obese.
The second station: the receptor was used to beat the mouse to test endotoxin to stimulate white blood cells to produce inflammatory factors, which first combined with leukocyte receptor CD14. If the CD14 is destroyed, white blood cells cannot sense endotoxins in the blood or produce inflammatory factors. It was found that when endotoxin was injected into CD14 knockout mice rabbit vibrator , these mice did not produce an inflammatory response and therefore did not develop obesity and insulin resistance.
The third station: the intestinal flora of mice was analyzed to determine the increase of antigens in the intestines and the way of entering the blood. the scientists analyzed the structure of intestinal flora of experimental mice by fluorescence in situ hybridization (fish). The results showed that the intestinal flora structure of obese mice fed with high-fat diet changed, the number of some beneficial bacteria (such as Bifidobacterium) decreased significantly, while the number of some Gram-negative bacteria increased significantly. This suggests that high-fat diet can increase the number of intestinal pathogenic bacteria, and then increase the amount of intestinal antigen.
A large number of studies have found that Bifidobacteria mainly grow on the intestinal mucosa, they can form a biofilm and have a protective effect on the intestinal tract. Once these bacteria are reduced, the intestinal wall will be damaged and the permeability will increase, which is what people call the intestines "leaking". Intestinal leakage, fecal poison into the blood, will naturally cause chronic inflammation, and eventually lead to chronic diseases.
Station 4: scientists who fed enough oligosaccharides to experimental mice added sufficient amounts of bifid factor (oligosaccharides) to a high-fat diet. Although the number of intestinal pathogens increased, because the number of beneficial bacteria did not decrease, antigens could not enter the bloodstream and could not stimulate leukocyte inflammation. This strong evidence suggests that an unreasonable diet destroys intestinal flora, causing chronic inflammation and chronic diseases.
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